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托瑞米芬 FARESTOS
托瑞米芬，Farestos (active agent - toremifene citrate) is a selective estrogen receptor modulator (SERM) derived from triphenylethylene. Toremifene was patented and approved in Europe in the mid 1990's to treat metastatic ER+ breast cancer in postmenopausal women.
In bodybuilding, toremifene citrate is used by men as a drug for post-cycle therapy (PCT) to counteract and/or block some of the effects of excessive estradiol in the body obtained through aromatization of various androgens used. Toremifene is used particularly to restore levels of endogenous testosterone in blood, as well as to prevent the estrogen-related side effect of gynecomastia.
描述
托瑞米芬Farestos (active agent - toremifene citrate) is a selective estrogen receptor modulator (SERM) derived from triphenylethylene. Toremifene was patented and approved in Europe in the mid 1990's to treat metastatic ER+ breast cancer in postmenopausal women.
In bodybuilding, toremifene citrate is used by men as a drug for post-cycle therapy (PCT) to counteract and/or block some of the effects of excessive estradiol in the body obtained through aromatization of various androgens used. Toremifene is used particularly to restore levels of endogenous testosterone in blood, as well as to prevent the estrogen-related side effect of gynecomastia.
Toremifene stimulates the hypothalamus which in turn stimulates the anterior pituitary gland to release gonadotrophic hormones. The gonadotrophic hormones are the follicle stimulating hormone (FSH) and luteinizing hormone (LH). FSH stimulates (in males) the spermatogenesis and LH stimulates the leydig cells of testicles to secrete more testosterone. This feedback mechanism is known as the hypothalamic-pituitary-testes axis (HPTA). This results in an increase of the body's own testosterone production causing blood levels to rise, as to compensate for the diminishing levels of exogenous steroids. This is vital to minimize post cycle muscle losses.
As for its antiestrogenic activity, it shall be noted that toremifene citrate, being a SERM, is a true estrogen blocker as compared to aromatase inhibitors. Toremifene citrate blocks the activity of estradiol at its target receptor sites in different tissues, and it does this by binding more strongly to the estradiol receptors in these tissues than estradiol itself, effectively taking the spot that estradiol normally would. Toremifene binds to the receptor site and remains inert (does not activate the receptors). This results in the inability of estradiol to bind to these receptors, as they have already been occupied by toremifene.
Toremifene does not lower overall circulating blood plasma levels of estradiol in the body, it merely blocks its activity in certain tissues. This is the main difference between aromatase inhibitors and Torem. AI’s bind to the aromatase enzyme responsible for the conversion of androgens into estradiol (aromatization), and reduce the total circulating levels of estradiol in the body by doing so.
Therefore, toremifene dosage, for a more effective boost in testosterone production, should be in the range of up to 120 mg per day. This dose can be maintained for a first week of PCT, 100 mg/day for the second week of PCT, and then 60 mg/day for the remaining 3 – 4 weeks of PCT. Of course, exact dosages shall be determined individually based on blood work results.
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